Assessing Whether Complements Role in Neurology Highlights a New Opportunity for Neurodegenerative & Neuropsychiatric Diseases

Time: 3:00 pm
day: Conference Day Two


  • Evidence from GWAS that structural variants of C4 confer a greater risk of developing schizophrenia
  • Using target genetic manipulation to mechanistically link C4 overexpression to aberrant circuit wiring
  • Utilising these techniques for the general the study of complement-associated neurological diseases such as age-related synapse elimination and Alzheimer’s disease models